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Compliance with Ethics Guidelines This article is based on previously conducted studies and does not contain any new studies with human participants or animals performed by the author

According to this hypothesis, glutamate dysregulation in OCD would be real, and potentially a useful therapeutic target, but would be a secondary consequence of neuronal hyperactivity that derived from some other pathophysiological process

However, in metabolically compromised animal models, elevated GH levels produced by GHRP-6 can induce transient insulin resistance in peripheral tissues a consideration relevant to studies involving metabolic disease models